![]() But it turns out that RA3 is pleiotropic, meaning that it influences more than one trait. It’s a reminder that it’s never too late to pursue our dreams. ![]() This surprised Klein and his colleagues, because RA3 was previously thought to play a role only in the way that plants branch. The Light of Hidden Flowers by Jennifer Handford Available NowAmazon. /rebates/2faudiobook2f2489962f&.com252faudiobook252f248996252f26afsrc3d126SID3d&idaudiobooks&ra4. The Light of Hidden Flowers is a deeply felt story of accepting who we are while pushing our boundaries to see how much more we can become. The result was that Klein and his coauthors discovered that another gene, RAMOSA3 (RA3), also plays a vital role in maize carpel programmed cell death. At the heart of their experiment is a technique known as an “forward genetics,” which is a way of working backwards from a known flower formation, or phenotype-in this case, a mutated maize flower caused by a genetic alteration in the cell death process-to the specific mutation causing that mutant formation.įinding the specific genes that govern any individual trait is like “finding a needle in a haystack,” according to Bartlett, and so Klein performed a series of genomic analyses with the complex maize genome. in plant biology at UMass Amherst and now at the Dana-Farber Cancer Institute, designed an innovative experiment to identify other genes regulating carpel suppression with GT1. Madelaine Bartlett in the maize field. Credit: UMass Amherstįocusing on maize flowers-think of an ear of corn or branched tassel at the top of a maize plant-the team, led by Harry Klein, who completed this research as part of his Ph.D. But it’s not the only gene regulating the process, and until now, no one has been able to identify others that interact with GT1 to suppress maize carpels. ![]() Enormous floral variety is governed by which parts of the flower’s growth are suppressed, and one gene in particular, known as GRASSY TILLERS1 (GT1), influences carpel growth suppression in maize. Programmed cell death is a genetic mechanism that eliminates some cells on purpose-it’s why humans don’t have webbed fingers-and it is at work in the carpels of flowers, or the seed-bearing structures at the heart of the flower. It has long been known that a process called “programmed cell death” is partly responsible for flowers’ morphological diversity. We want to know how the same few parts wind up creating such wildly different forms.” “They’re all built of the same component parts, and yet we have such incredible diversity, from corn tassels to orchids. “Flowers are amazing,” says Madelaine Bartlett, professor of biology at UMass Amherst and senior author of the paper, recently published in the Proceedings of the National Academy of Sciences. Mutant maize tassels in which programmed cell death has been suppressed. Credit: UMass Amherst
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